Ħażin nifs u Helicobacter pylori
Kif għamel in-nifs come about? Not only għax the simple oral cleaning is not in place, but also għax of the Helicobacter pylori in the stomach!
Huwa il biss super batterji li jista ' jeżisti fi l- stonku , u it is ma jibża ' ta' b'saħħtu stonku aċidu ... Il- ħażin nifs it kawżi ma jistax ikun meħlus anke minn tfarfir snien Nies b' obvious ton għandu jagħti prijorità lil jekk huma infettati ma ' Helicobacter pylori!
Ħafna nies huma at a telf meta ffaċċjat ma it-test riżultati ta' Helicobacter pylori. So, is there any effective way to deal with this bacterium moħbi in the junction between the end of the stomach and the intestinal tract?
Il- most direct method is antibiotic treatment, among li amoxicillin, clarithromycin, levofloxacin, metronidazole, tetracycline and other antibiotics are commonly used drugs for the treatment of Helicobacter pylori. Madankollu, għax of the serious abuse of antibiotics, especially amoxicillin, levofloxacin and metronidazole, the human body has a very high resistance to these drugs, and it is easy to cause intestinal flora imbalance, so the simple antibiotic treatment in the past It is no longer applicable.
Il mediku komunità dawwar its ġdid tama ta ' trażżin Helicobacter pylori għal probijotiċi. Esperimenti urew li a varjetà ta' probijotiċi tali bħala bifidobacteria, lactobacilli u ħmira can jinibixxi għal a ċertu firxa , u hemm aktar minn wieħed mod u mod għal probijotiċi biex jinibixxu Helicobacter pylori.
kompetittiv adeżjoni
Helicobacter pylori prinċipalment jaderixxi ma' l-epiteljali ċelloli ta' l-gastriku mukoża u jipproduċi tossini u ta' ħsara sustanzi - ammonja. Madankollu, probijotiċi jista' reduce il possibbiltà ta' Helicobacter pylori adeżjoni to epiteljali ċelloli through the "snatching" effect, li is, li by competiing with Helicobacter pylori for the mucosal epithelial cell binding site.
anti-infjammatorju effett
Cytotoxin-encoded protein is an important factor leading to Helicobacter pylori infection. Experiments have found that Lactobacillus salivarius can inhibit the expression of the encoded protein, thereby reducing the inflammatory response of the gastric mucosa.
Fi addition, probiotics may also regulate the body's immune response, such as stimulating the intestinal mucosa to secrete immunoglobulins, enhance the body's resistance to the toxins produced by Helicobacter pylori, and enhance the intestinal barrier function.
Some bacteriocins, organic acids (including lactic acid, formic acid, acetic acid, propionic acid, etc.) u short-chain fatty acids produced by probiotics can inhibit or even kill Helicobacter pylori. Studies have shown that Bacillus subtilis, Bifidobacterium, Enterococcus faecalis and Lactobacillus can all release bacteriocins to disrupt the colonization and development of Helicobacter pylori.
lactobacillus plantarum HH-LP78
Patents: CN114259055A